File:Free-Fatty-Acid-Induced-PP2A-Hyperactivity-Selectively-Impairs-Hepatic-Insulin-Action-on-Glucose-pone.0027424.g003.jpg
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editDescriptionFree-Fatty-Acid-Induced-PP2A-Hyperactivity-Selectively-Impairs-Hepatic-Insulin-Action-on-Glucose-pone.0027424.g003.jpg |
English: □ Control; ▪ 0.5 mM Palmitate. However, insulin action on TG accumulation is maintained. (a) Insulin-stimulated suppression of gluconeogenesis (from medium containing 2 mM pyruvate), (b) incorporation of glucose into glycogen (from 5.5 mM glucose) and (c) glucose disposal (from 15.5 mM glucose) were significantly inhibited in hepatocytes treated for 20 h with palmitate in vitro. Meanwhile, insulin-stimulated TG accumulation was maintained in palmitate-treated hepatocytes (d). Results are expressed as differences from the rates in cells given no insulin. Data are averages of 8–10 independent experiments +/− std. error of the mean. * indicates p<0.05 between groups. |
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Source | Image file from Galbo T, Olsen G, Quistorff B, Nishimura E (2011). "Free Fatty Acid-Induced PP2A Hyperactivity Selectively Impairs Hepatic Insulin Action on Glucose Metabolism". PLOS ONE. DOI:10.1371/journal.pone.0027424. PMID 22087313. PMC: 3210172. | |
Author | Galbo T, Olsen G, Quistorff B, Nishimura E | |
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current | 22:42, 7 May 2016 | ![]() | 563 × 339 (66 KB) | Recitation-bot (talk | contribs) | Automatic upload of media from: doi:10.1371/journal.pone.0027424 |
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