Identifier: 39002086343333.med.yale.edu
Title: A treatise on mental diseases
Year: 1900 (1900s)
Authors: Berkley, Henry Johns,1860-
Subjects: Insanity Mental Disorders
Publisher: New York, D. Appleton & Company
Contributing Library: Yale University, Cushing/Whitney Medical Library
Digitizing Sponsor: Open Knowledge Commons and Yale University, Cushing/Whitney Medical Library

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majority,are induced directly by the insufficiency of the supply of nourish-ment to the cell body, others by the irritant qualities of a virus per-meating from the blood channels, as in syphilis or chemical intoxi-cations. In conclusion, it may be said that the pathology of thenucleus is far more definite than that of the protoplasm, and whileat the present time we may not be able to correctly interpret themeaning of the lesion, we are at least able to recognise when themost important portion of the cell structure is involved in a degen-erative process. The staining of the entire neurone by the chrome-silver methodof Golgi should apparently offer a splendid field for pathologicalresearch. But while this is entirely true for experimental work,unfortunately, when we come to the brain of man, we are againhampered by factors with which we do not have to deal in thelower animals—the presence of post-mortem products, and thedifficulty in obtaining fair staining of the preparations. Again,
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Fig. 11.—Description of the Drawings. No. 1. Psychical cell from the second cellular layer ofthe cortex, showing a few pathological tumefactions on the uppermost brandies of the apical den-drite. Otherwise the cell is normal. Bicin poisoning of 36 hours1 duration. Subcutaneous injec-tion of 0.5 milligramme ricin. Compare frontispiece. No. 2. Projection cell from the secondlayer of the- cortex, showing an increased number of pathological swellings on the finer stems ofthe cell. There is now distinct diminution of the gemmulse wherever the swellings are found.Ricin poisoning of 48 hours duration. Subcutaneous injection of 0.25 tag. ricin. No. 3. Projec-tion cell of the lung apical process variety, showing numbers of large swellings of the protoplasmof the apical dendrite, thinning of the protoplasm of the stems in the interval between the nod-ules, and considerable loss of the gemmulse along the margins. The lateral branches have mainlydisappeared. The basal processes are retained int

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