Identifier: cu31924023990744 (find matches)
Title: The diseases of China : including Formosa and Korea
Year: 1910 (1910s)
Authors: Jefferys, W. Hamilton (William Hamilton), 1871-1945 Maxwell, James L
Subjects:
Publisher: London : Bale & Danielsson
Contributing Library: Cornell University Library
Digitizing Sponsor: MSN

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normous amount of very strong evidence has been collected; indeed, so strcjng that listening to it one feels absolutely convinced of its truth. But these defenders of the rice theory, despite all the evidence in their favour, have failed utterly to rel)ut other evidence, which to the outside observer makes that theory untenable. Tliis is the as_\-lum Ijeri-beri of non-rice-eating countries, the occurrence of epidemic beri-beri in non-rice-eating regions, such as parts of South America, and the beri-beri found on ships where rice takes no part in the menu, and of which we have convincinig, testimony from the most skilled experts that the disease is indistinguishable from true beri-beri. Then when we again come to the believers in the rice origin, we haw the inirst striking differences in their theories, each observer supporting his own with indisputable statistics! Take for example: Braddon believes that unciircfl rice (cured rice being rice ))arboile(l ininudiati l\- it is liar-
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CLINICAL PICTURE OF BERI-BERI. lOO vested) is the cause of beri-beri, and brings to bear a number of most striking figures. Unfortunately, he omits the figures where the use of cured rice has not proved a certain preventive of the disease. On the other hand, we have Vordermans statistics from Java, which would show by equally good figures that beri-beri is due to the consumption of husked rice, those who eat the rice in the husk (and therefore all the toxins) escaping the disease. About these theories, it must be said that the latter failed to stand the test of time, and the former has had, as yet, no opportunity of doing so. Hamilton Wright has promulgated the theory that the disease is atoxEemia resembling diphtheria, the primary lesion being in the stomachand duodenum, where the germ which he describes as causal generates its toxin and produces a gastro-duodenitis. The source of the infection he believes to be the excreta of beri-beri patients. Experimental post-mortem investigation in Ho

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